10 Ways to Write More Effective Ads by anonymous

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This is exemplified by thiopurine S-methyltransferase (TPMT) polymorphisms and azathioprine (AZA)/ 6-mercaptopurine (6-MP) toxicity. The therapeutically active metabolites of these compounds are the 6-thioguanine nucleotides (6-TGNs) whose production is limited by xanthine oxidase and TPMT. Xanthine oxidase is absent in haematological tissue and therefore there is no secondary pathway to prevent high levels of myelotoxic 6-TGNs if the TPMT pathway is defective. Several TPMT variants that cause reduced enzymatic activity have been identified of which TPMT∗ 2 and TPMT∗ 3A are the most common [150–152].

Specific enzymatic amplification of DNA in vitro: the polymerase chain reaction. Cold Spring Harb Symp Quant Biol 1986; 51(Pt 1):263–73. 2 Weissenbach J, Gyapay G, Dib C, et al. A secondgeneration linkage map of the human genome. Nature 1992;359(6398):794–801. 3 RK Russel JS. IBD: a family affair. Best Pract Res Clin Gastroenterol 2004;18(3):525–39. 4 Yang H, McElree C, Roth MP, et al. Familial empirical risks for inflammatory bowel disease: differences between Jews and non-Jews. Gut 1993;34(4):517–24.

Despite a variety of genotype/phenotype associations within the HLA region the lack of consistency and relatively low allele frequencies of disease-associated mutations are at present a barrier to any clinical application. 24 CHAPTER 2 ................................................................................................................................................................................................................................... Pharmacogenetics A field that does show some promise in current clinical practice is that of pharmacogenetics.

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